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ToxCard: Second Generation Antipsychotic Overdose

EMDocs

of exposures reported to poison control centers in 2021 were related to antipsychotics or sedative-hypnotics with the majority of those exposures occurring in patients 20 years of age or older. Consult a medical toxicologist or regional poison control (at 1-800-222-1222 in the United States). 1 Class IA (e.g., procainamide), IC (e.g.,

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Case Report: A Child with a Rash and Lead Poisoning History

ACEP Now

On physical examination, the child was non-toxic, well-nourished, alert, awake, and not in acute distress. Incidentally, the day after ED visit number one, the mother was called by the child’s pediatrician advising her to bring him for evaluation of lead poisoning. Discussion Lead poisoning is a serious health concern.

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Chemical Burns

Mind The Bleep

Alkali burns result in liquefaction necrosis, allowing for deeper tissue injury as well as vascular injury that can lead to both local and systemic toxicity [1]. Circulation Assess heart rate, blood pressure, peripheral and central CRT, pulses and 3 lead ECG. Establish IV access and begin fluid resuscitation with 250ml boluses of 0.9%

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A comatose patient with a carbon monoxide level over 50%

Dr. Smith's ECG Blog

An ECG is always recorded for CO toxicity. This was his ECG. Intravascular Neutrophil Activation Due to Carbon Monoxide Poisoning What do you think of this ECG? The assessment of QT intervals in acute carbon monoxide poisoning I was not worried about this ECG. There is no reciprocal ST depression in aVL.

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Beyond the Burns: Toxic House Fire Gases

Northwestern EM Blog

The post goes into detail on the pathophysiology, signs, symptoms, and diagnosis of both carbon monoxide and cyanide poisonings, so this commentary will focus on the clinical approach. An elevated lactic acid is a surrogate for cyanide poisoning, specifically a level of 8-10 mmol/L or greater is sensitive and should prompt intervention.

Burns 52
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Diffuse Subendocardial Ischemia on the ECG. Left main? 3-vessel disease? No!

Dr. Smith's ECG Blog

An immediate 12-lead EKG was obtained: There is ST elevation in leads aVR and V1, with marked ST depression in I, II, III, aVF, V3-V6. DISCUSSION: The 12-lead EKG EMS initially obtained for this patient showed severe ischemia, with profound "infero-lateral" ST depression and reciprocal ST elevation in lead aVR. What should be done?

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A guide to passing MRCP Part 2 written

Mind The Bleep

Therefore, I’m sharing the things that worked well and were time-effective, and those that didn’t. Many questions were repeated, and on some, the resources were not included (“look at this ECG” but no ECG was visible). Other resources Flashcards worked very well for me, but aren’t everyone’s cup of tea. Survivorship bias.

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