Remove 2015 Remove Hyperthermia / Hypothermia Remove Shock
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Chemical Burns

Mind The Bleep

Sodium Chloride or Hartmanns if indicated, monitoring for signs of shock. Exposure Expose the patient in a systematic manner while keeping remaining body areas covered e.g. 1 limb at a time, to reduce the risk of hypothermia. 2015 Jun;11(2):227-31. Establish IV access and begin fluid resuscitation with 250ml boluses of 0.9%

Burns 52
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The Science on Targeted Temperature Management

ACEP Now

Early work on TTM in 2002 showed benefit to cooling to 33 degrees Celsius, which subsequently influenced international resuscitation guidelines to recommend mild hypothermia at 32 degrees to 34 degrees Celsius in 2005. Adding nuance, this study also showed that the highest-severity arrests did not benefit from hypothermia.

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EM@3AM: Hyperthermia

EMDocs

Heat stroke can lead to end-organ dysfunction such as rhabdomyolysis, disseminated intravascular coagulation, cardiogenic shock, liver failure, and cerebral edema. Broad-spectrum antibiotics (A) for septic shock are not inappropriate given the hyperthermia, tachycardia, and hypotension. Hyperthermia. Heatstroke.

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Acute Hyperthermia in the Emergency Department

Taming the SRU

J Intensive Care Med 2015. First aid cooling techniques for heat stroke and exertional hyperthermia: A systematic review and meta-analysis. Classification of sepsis, severe sepsis and septic shock: the impact of minor variations in data capture and definition of SIRS criteria. References Chiha M, Samarasinghe S, Kabaker AS.

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Vasopressor Nonresponse

Northwestern EM Blog

Written by: Elizabeth Stulpin, MD (NUEM ‘23) Edited by: Aaron Wibberly, MD (NUEM ‘22) Expert Commentary by : Joshua Zimmerman, MD (NUEM ‘17) Non-Response to Vasopressors Shock is defined as a state of cellular and tissue hypoxia resulting in end organ dysfunction. And for most forms, EM physicians are not typically shocked by shock.

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Penetrating chest trauma

Don't Forget the Bubbles

Ranulf went white with the shock. Traditionally thought to result from the dilutional effects of massive transfusion, acidaemia or hypothermia, trauma-induced coagulopathy is now known to be triggered at the time of injury, long before these factors have developed or any medical intervention has taken place. 2015 Jul;115(1):76-83.

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The EMERGE Trial: Emergency vs Delayed Catheterization in Survivors of Out-of-Hospital Cardiac Arrest

RebelEM

Background: Previous observational studies published in 2015 (Geri 2015)(Vyas 2015) indicated that early cardiac catheterization in patients with out-of-hospital cardiac arrest (OHCA) might improve mortality and result in more favorable neurological outcomes. Cerebral unresponsiveness CPC 5: Brain death. Bougouin, W.,